We were delighted to host his excellency the Head of the ICRC for the Gulf Cooperation Council
@MamadouSowICRC
at the Saudi Red Crescent Volunteer Center in Medina. During his visit, we demonstrated the work of the volunteer team and highlighted the capabilities of the center. He
Just attended and presented at the 9th SASEM Conference 2024. It was an amazing experience with experts from around the globe discussing important topics in Emergency Medicine. Huge thanks to the organisers and scientific committee for putting together such a fantastic
Prehospital Blood Administration, Are We There Yet?
By Dr. Ahmed Ghabban
“Among the various causes of shock in patients, prehospital hemorrhagic shock stands out as particularly challenging to manage.”
“In conflict zones, getting to the hospital during evacuation and transit
A DM patient presented with 2 hours of right shoulder pain. While finishing an ECG, the patient arrested (Vfib) and was successfully defibrillated. Has a GCS of 15/15. Underwent PPCI (LMCA).
Take-home points:
- Always be suspicious of atypical chest pain, especially in diabetic
@ecgandrhythmRoe
Bifascicular block + first degree heart block. (Previously called incomplete trifascicular block). Not an indication for pacing, usually.
If it’s true trifascicular block it should be associated with third degree AV block and Pt will require pacemaker insertion.
@DrRazi4
@PMcardioBot
@smithECGBlog
@drharikrishrau
******Take Home points****** :
Don’t ever forget aVL lead. ST depression in aVL can be a marker of serious problems :
- Significant mid LAD lesion.
- Evolving inferior MI and possible RV involvement.
@DrRazi4
@PMcardioBot
Yes, very suspicious OMI inferior leads with subtle ST depression Avl. I will add that’s not first degree AV block but I think it’s second degree AV block 2:1.
@Dr_MohammedGh
It was truly a world class event! Huge thanks to you, Dr. Mohammad ,Dr. Faisal and your team, for your dedication to reaching this level of excellence.
@Faisal_Bk
@Dr_MohammedGh
@DrRazi4
@drharikrishrau
@PMcardioBot
Looks like sinus bradycardia, Wenckebach, and if he doesn’t take any beta-blockers, calcium channel blockers, digoxin, and amiodarone. Treatment is rarely required.
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@ecgandrhythmRoe
Ventricular rate around 36!
RBBB with LAFB.
Right ventricular strain pattern as T-wave inversions are seen in the precordial leads (V3-V6) and inferior leads (II, III and aVF).
?? V1 looks like atrial flutter with low responding ventricular.
@ecgandrhythmRoe
My analysis: heart rate around 95, incomplete RBBB, 2 premature complexes, P wave shape suspected of atrial enlargement. also there’re 3 different P wave morphology?mat.
@saeedalsuhaimi
لافض فوك اخي سعيد ،واحتياطاً عند وجود الأعراض التالية يجب الذهاب الى قسم الطوارئ:
١- منطقة العضة تكون ساخنة عند اللمس ومنتفخة.
٢- ارتفاع في درجة حرارة الجسم.
٣- اعراض الصدمة التحسسية (ضيق في التنفس،انتفاخ اللسان او مجرى الهواء، احمرار الجلد، الدوخه، غياب الوعي…)
ولا ننسى
@ecgandrhythmRoe
ECG: Sinus tachycardia, normal axis, likely there is Rt atrial enlargement and unifocal Trigeminy (every third beat is a PVC and they’re identical)
Holter: NSVT.
@ecgandrhythmRoe
Sinus rhythm,normal axis, kind of LAE P wave shape, T wave in inferior leads are the same size as the QRSs! Not that clear if there’s subtle ST depression in Avl. I would do more Hx, ECHO...
it looks suspicious!
@ecgandrhythmRoe
My analysis for this ECG: Afib with rapid ventricular response and QRS morphology of LBBB, heart rate about 116, and seems there is accessory pathway (delta wave v5,v6) , QRS voltage variable throughout the ECG maybe because of pericardial effusion?
@smithECGBlog
Very cool and beautiful scientific paper and its results are clear in favor of OMI.
When do you expect practice to change globally from STEMI to OMI?
New clinical policy from ACEP on management of emergency
department patients presenting with severe agitation:
1- (Best): Use a combination of droperidol and midazolam.
2- (Second Best): If droperidol not available use atypical antipsychotic