Nixon Lab at the CDR
@cdrnixonlab
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Link to Nature Neuroscience paper - https://t.co/oXSUCOyHuI
Joined April 2022
New paper out in @elife. mTOR inhibition in Q175 Huntington’s disease model mice facilitates neuronal autophagy and mutant huntingtin clearance This study presents a valuable finding on the alterations in the autophagic-lysosomal pathway in a HD model.
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Members from Nixon Lab contributed to a paper titled "Pathophysiologic abnormalities in transgenic mice carrying the Alzheimer disease PSEN1 Δ440 mutation" from Monteiro Lab.
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New review paper by Dr. Ralph Nixon "Autophagy–lysosomal-associated neuronal death in neurodegenerative disease".
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We are excited to announce that Dr. Ralph Nixon, with Dr. David Rubinsztein, have published a review paper titled, “Mechanisms of autophagy–lysosome dysfunction in neurodegenerative diseases," in the Aug 2024 issue of Nature Reviews Molecular Cell Biology.
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RT @alzassociation: Breaking News: Eating about two servings per week of processed red meat—like bacon, bologna and salami—could raise deme…
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Congratulations to Ralph A. Nixon for receiving the Khalid Iqbal Lifetime Achievement Award #AAIC24 honoring his contributions and career-long commitment for Alzheimer's and dementia research.
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RT @alzassociation: Congratulations to Ralph A. Nixon, M.D., Ph.D., the recipient of the Khalid Iqbal Lifetime Achievement Award. Dr. Nixon…
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We are hiring!! We are seeking a Research Scientist in Bioinformatics and Computer Biology. For more details and to apply
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RT @marc_herb: @cdrnixonlab @biorxivpreprint A Comprehensive Enumeration of the Human Proteostasis Network. 2. Components of the Autophagy…
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Findings of the study from the Nixon Lab is highlighted in Newsweek Magazine: "New Alzheimer's Drug Brings Hope—and Tough Choices for Patients, Caregivers." Follow the link to find more -
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Thrilled to announce that the work from the Nixon Lab is highlighted in Quanta Magazine: "What Causes Alzheimer’s? Scientists Are Rethinking the Answer." Follow the link to find more -
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The authors show in an animal model and in a study in patients with dementia with Lewy bodies (DLB) that the drug neflamapimod has potential to treat diseases, such as DLB, associated with loss of neurons that produce the neurotransmitter acetylcholine.
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Taken together, the findings indicate that neflamapimod appears to represent a scientific breakthrough towards treating degeneration of the basal forebrain cholinergic system, a long-standing objective of drug development for a range of neurodegenerative diseases. (n/n)
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