![Agingdoc⭐David Barzilai🔔MD PhD MS MBA DipABLM🩺 Profile](https://pbs.twimg.com/profile_images/1604280159154077696/O7ZBZUOH_x96.jpg)
Agingdoc⭐David Barzilai🔔MD PhD MS MBA DipABLM🩺
@agingdoc1
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Longevity medicine best practices | healthspan | precision medicine | bio/acc | consulting @ https://t.co/XWGLGiSPyZ Connect: https://t.co/wneqWlNlzd
Geroscience & Aging Biology 🌎
Joined May 2020
Thank you @hevolution_f , your vision and dedication to extending human health is truly inspiring. As a global community, we are driving proactive approaches to advancing healthspan worldwide. 🌍 🔗
I’m delighted to be speaking at the Global Healthspan Summit: Architecting the Future, hosted by the Hevolution Foundation—the premier global gathering advancing longevity science and translating breakthrough research into improved healthspan outcomes. Looking forward to a day of insightful discussions and to helping shape the future of healthspan! 👨⚕️ 🔗| @hevolution_f
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@sabrinablittle @davidasinclair You and David both have fine points. IMHO, oversized is in the eye of the beholder so it’s hard to judge from the outside what the right balance should be for another. I see where to draw the line on health-focus as a personal choice… though far too many neglect it entirely. 👨🏻⚕️
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RT @agingdoc1: Precision Longevity Medicine With Dr. David Barzilai MD PhD ⚕️ @InsideTracker and @agingdoc1 🔗 ht…
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Polyphenols and Microbiota Modulation: Insights from Swine and Other Animal Models for Human Therapeutic Strategies @Molecules_MDPI
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Ageing and inflammation limit the induction of SARS-CoV-2-specific CD8+ T cell responses in severe COVID-19 @JCI_insight
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Longitudinal serum proteome mapping reveals biomarkers for healthy ageing and related cardiometabolic diseases 🔑"Our data constitute a valuable resource and offer useful insights into the roles of serum proteins in ageing and age-associated cardiometabolic diseases, providing potential targets for intervention with therapeutics to promote healthy ageing." | @NatMetabolism
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mTOR signalling controls protein aggregation during heat stress and cellular aging in a translation- and Hsf1-independent manner "we found that reduced mTOR activity, achieved through deletion of TCO89, an mTORC1 subunit, almost completely prevents protein aggregation during heat stress and aging without reducing global translation rates..."
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